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Michael Smith Laboratories/Botany
University of British Columbia, Canada
Office phone: +1-604-822-3155
Lab phone: +1-604-822-3205
E-mail: xinli@mail.ubc.ca
https://botany.ubc.ca/people/xin-li
Area of expertise: Plant immunity; Resistance protein signaling; NLR receptors; Arabidopsis molecular genetics; E3 ubiquitin ligases; SCF
Selected Publications:
Peng, Y., Yang, J., Li, X., and Zhang, Y. (2021). Salicylic acid: Biosynthesis and Signaling. Annu. Rev. Plant Biol. doi:10.1146/annurev-arplant-081320-092855.
Wu, Z., Huang, S., Zhang, X., Wu, D., Xia, S., and Li, X. (2017). Regulation of plant immune receptor accumulation through translational repression by a glycine-tyrosine-phenylalanine (GYF) domain protein. eLife
Johnson, K.C., Zhao, J., Wu, Z., Roth, C., Lipka, V., Wiermer, M., and Li, X. (2017). The putative kinase substrate MUSE7 negatively impacts the accumulation of NLR proteins. Plant J. 89: 1174–1183.
Ding, P., Rekhter, D., Ding, Y., Feussner, K., Busta, L., Haroth, S., Xu, S., Li, X., Jetter, R., Feussner, I., and Zhang, Y. (2016). Characterization of a pipecolic acid biosynthesis pathway required for systemic acquired resistance. Plant Cell 28: 2603–2615.
Huang, S., Balgi, A., Pan, Y., Li, M., Zhang, X., Du, L., Zhou, M., Roberge, M., and Li, X. (2016). Identification of methylosome components as negative regulators of plant immunity using chemical genetics. Mol. Plant 9: 1620–1633.
Copeland, C., Woloshen, V., Huang, Y., and Li, X. (2016). AtCDC48A is involved in the turnover of an NLR immune receptor. Plant J. 88: 294–305.
Johnson, K.C.M., Yu, Y., Gao, L., Eng, R.C., Wasteneys, G.O., Chen, X., and Li, X. (2016). A partial loss-of-function mutation in an Arabidopsis RNA polymerase III subunit leads to pleiotropic defects. J. Exp. Bot. 67: 2219–2230.
Dong, O.X., Tong, M., Bonardi, V., Kasmi, F.E., Woloshen, V., Wünsch, L.K., Dang, J.L., and Li, X. (2016). TNL-mediated immunity in Arabidopsis requires complex transcriptional regulation of the redundant ADR1 gene family. New Phytol. 210: 960–973.
Li, X., and Zhang, Y. (2016). Suppressor screens in Arabidopsis. Methods Mol. Biol. 1363:1–8.
Huang, S., Chen, X., Zhong, X., Li, M., Ao, K., Huang, J., and Li, X. (2016). Two redundant plant TRAF proteins participate in NLR immune receptor turnover. Cell Host Microbe. 19: 204–215.
Dong, O.X., Meteignier, L.V., Plourde, M.B., Ahmed, B., Wang, M., Jensen, C., Jin, H., Moffett, P., Li, X., and Germain, H. (2015). Arabidopsis TAF15b localizes to RNA processing bodies and contributes to snc1-mediated autoimmunity. Mol. Plant Microbe Interact. 29: 247–257.
Disch, E.M., Tong, M., Kotur, T., Koch, G., Wolf, C.A., Li, X., and Hoth, S. (2015). Membrane-associated ubiquitin ligase SAUL1 suppresses temperature- and humidity-dependent autoimmunity in Arabidopsis. Mol. Plant Microbe Interact. 29: 69–80.
Kapos. P., Xu. F., Meinnel. T., Giglione. C., and Li, X. (2015). N-terminal modifications contribute to flowering time and immune response regulations. Plant Signal. Behav. 10: e1073874.
Johnson, K.C., Xia, S., Feng, X., and Li, X. (2015). The chromatin remodeler SPLAYED negatively regulates SNC1-mediated immunity. Plant Cell Physiol. 56:1616–1623.
Li, X., Kapos, P., and Zhang, Y. (2015). NLRs in plants. Curr. Opin. Immunol. 32C:114–121.
Xu, F., Huang, Y., Li, L., Gannon, P., Linster, E., Huber, M., Kapos, P., Bienvenut, W., Polevoda, B., Meinnel, T., Hell, R., Giglione, C., Zhang, Y., Wirtz, M., Chen, S., and Li, X. (2015). Two N-terminal acetyltransferases antagonistically regulate the stability of a nod-like receptor in Arabidopsis. Plant Cell 27: 1547–1562.
Xu, F., Zhu, C., Cevik, V., Johnson, K., Liu, Y., Sohn, K., Jones, J.D., Holub, E.B., and Li, X. (2015). Autoimmunity conferred by chs3-2D relies on CSA1, its adjacent TNL-encoding neighbour. Sci. Rep. 5: 8792.
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